August 2022 Br J Cardiol 2022;29:119–20 doi:10.5837/bjc.2022.030
Manuel Felipe Cáceres-Acosta, Bairon Díaz Idrobo, Diana Carolina Urbano Albán
Introduction Since its appearance in December 2019 in Wuhan (Hubei, China), more than 300 million people worldwide have been infected with the SARS-CoV-2 virus1 (which generates multi-systemic involvement and is an emerging cause of myocardial compromise). This article presents an illustrative case of dilated cardiomyopathy as a complication of viral myocarditis due to SARS-CoV-2. Case presentation Table 1. Most relevant laboratory findings Test Result Reference values Leukocytes 13,400/µL 4,200–10,400/µL Lymphocytes 1,300/µL 300–1,500/µL Haemoglobin 14 g/dL 11.7–15.5 g/dL Platelets 282,000/µL 156,000–373,000/µL
July 2022 Br J Cardiol 2022;29:117–8 doi:10.5837/bjc.2022.026
Federico Liberman, Roberto Cooke, María J Cabrera, Santiago Vigo, Guillermo Allende, Luciana Auad, Juan P Ricarte-Bratti
Introduction High-output heart failure (HF) is an uncommon condition with varying aetiologies, often left uninvestigated. This type of HF is characterised by an elevated cardiac output, usually with high stroke volume, leading to biventricular dilation. According to a Mayo Clinic series involving 120 patients with high-output HF diagnosed between 2000 and 2014, the most important causes included obesity, liver cirrhosis, chronic obstructive pulmonary disease, and shunting, such as fistulas or arteriovenous malformations.1 Case report A 30-year-old man presented to the emergency department for dyspnoea, abdominal distention, and oedema lasting
July 2004 Br J Cardiol (Acute Interv Cardiol) 2004;11:AIC 70–AIC 72
Mark J Earley, Michael AJ Park, Richard J Schilling
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