Introduction
Retinal ischaemic perivascular lesions (RIPL) describe characteristic atrophic changes of the inner nuclear layer (INL) as a consequence of ischaemic injury.1 These are illustrated by spectral domain optical coherence tomography (SD-OCT) as focal thinning of the retinal INL, contemporaneous with enhanced hypo-reflectivity of the outer nuclear layer (ONL).2 RIPLs are chronic lesions originating from paracentral acute middle maculopathy (PAMM); these are hyperacute ischaemic lesions of the middle retina and are associated with multifarious vascular diseases.3,4 As opposed to their hyperacute predecessors (PAMMs), which resolve with
